A peptide that selectively disrupts a pathological α2δ-1 protein–protein interaction can reduce mechanical hypersensitivity in lumbosacral radiculopathy, without the sedation and dependence liabilities that limit gabapentinoids.

A peptide that selectively disrupts a pathological α2δ-1 protein–protein interaction can reduce mechanical hypersensitivity in lumbosacral radiculopathy, without the sedation and dependence liabilities that limit gabapentinoids. | OpenLabs